The study identified resistance mechanisms in Neisseria meningitidis, including penA gene polymorphisms associated with penicillin and ampicillin resistance, catP gene-mediated chloramphenicol resistance, folP gene mutations linked to sulfisoxazole and trimethoprim-sulfamethoxazole resistance, tet(B) efflux pump contributing to tetracycline resistance, and rpoB gene mutations causing rifampin resistance. Additionally, gyrA mutations were associated with reduced fluoroquinolone susceptibility.

The study identified the catP gene as responsible for chloramphenicol resistance in Neisseria meningitidis isolates from military camps in Vietnam. Additionally, the mtrR gene with the A39T mutation was found to potentially increase antibiotic resistance.

This study identifies multiple AMR genes and mutations in Clostridioides difficile, including ermB, tetM, mefH, and various mutations in gyrA, gyrB, rpoB, pbp1, and pbp3. These findings highlight the widespread nature of AMR in C. difficile and its potential role in the spread of the bacterium.

The study identified several AMR genes and mutations in Clostridioides difficile isolates, including ermB conferring resistance to MLSB antibiotics, catP conferring resistance to chloramphenicol, and mutations in gyrA, gyrB, and rpoB that confer resistance to fluoroquinolones and rifamycins.

The study identified several AMR genes and mutations in C. difficile isolates from Mexico, including cfrE, ermB, aadE, aadA27, aac(6')-Ie-aph(2'')-Ia, catP, blaCDD2, blaCCD1, tetM, tetO, tetB, tetA, and cdeA, along with mutations in rpoB, rpoC, fusA, and pbp2 associated with resistance to various antibiotics.

The study identifies numerous AMR genes in Mycobacteroides abscessus complex, highlighting the widespread presence of resistance to multiple antibiotic classes, including beta-lactams, aminoglycosides, glycopeptides, and others. Key findings include the detection of beta-lactamases like blaLAP-1 and blaTLA-2, 23S rRNA methyltransferases such as erm(33), erm(43), and erm(44), and various aminoglycoside modifying enzymes. Additionally, vancomycin resistance genes like vanA, vanB, and vanC were identified, along with efflux pump genes contributing to multidrug resistance.

The study identified novel plasmid families in Clostridium perfringens, including a novel conjugative locus (Bcp) and several antimicrobial resistance genes such as tetA(P) and tetB(P).

The study identifies the chloramphenicol-resistant ST-1576 lineage of Neisseria meningitidis in Southern Vietnam, highlighting the presence of the catP gene encoding chloramphenicol acetyltransferase and mutations in gyrA contributing to ciprofloxacin resistance.

The study identified multiple antimicrobial resistance genes in Avibacterium paragallinarum isolates from Guangdong, China, including tet(B), aph(3')-Ia, aph(3'')-Ib, aph(6)-Id, aad-A, sul2, sul3, catP, floR, blaOXA-1, blaCTX-M-14, erm(X), mef(B), and fosA3. These genes were associated with resistance to tetracycline, streptomycin, kanamycin, trimethoprim-sulfamethoxazole, chloramphenicol, ampicillin, erythromycin, and fosfomycin.