Browse AMR Genes
Explore antimicrobial resistance genes from the literature
Explore antimicrobial resistance genes from the literature
heavy metal efflux and resistance gene
Overview
Complete genome sequence and phenotype microarray analysis of Cronobacter sakazakii SP291: a persistent isolate cultured from a powdered infant formula production facility.
The study identified several AMR genes in Cronobacter sakazakii SP291, including tellurite resistance genes (terX, terW, terA, terB, terC, terD), heavy metal efflux genes (cusS, cusR, cusC, cusF, czcB, czcA, cusA, copG, pcoS, pcoB, pcoA), and oxidative stress resistance genes (yebA, yibP, hmp, grxB, grxC).
Silver resistance in Gram-negative bacteria: a dissection of endogenous and exogenous mechanisms.
The study identifies two point mutations in ompR and cusS that confer endogenous silver resistance in E. coli, and highlights the role of the silE gene in exogenous silver resistance.
Comparative genomic and phenotypic characterization of invasive non-typhoidal Salmonella isolates from Siaya, Kenya.
The study identified several AMR genes in Salmonella isolates from Kenya, including blaTEM-1, aadA1, strA, strB, catA1, dhfr1, sul1, and sul2, which confer resistance to various antibiotics such as penicillins, cephalosporins, streptomycin, chloramphenicol, trimethoprim, and sulfonamides.
Mutations in SilS and CusS/OmpC represent different routes to achieve high level silver ion tolerance in Klebsiella pneumoniae.
The study identifies mutations in silS, cusS, and ompC as key mechanisms for achieving high-level silver ion tolerance in Klebsiella pneumoniae. These mutations lead to overexpression of the silCFBA or cusCFBA operons and disruption of the outer membrane porin OmpC, resulting in increased silver tolerance.
Study of heavy metal resistance genes in Escherichia coli isolates from a marine ecosystem with a history of environmental pollution (arsenic, cadmium, copper, and mercury).
The study identified several heavy metal resistance genes (HMRGs) in Escherichia coli isolates from a marine ecosystem with a history of environmental pollution, highlighting the prevalence of these genes and their potential interactions with antibiotic resistance genes.
It Takes Two to Make a Thing Go Right: Epistasis, Two-Component Response Systems, and Bacterial Adaptation.
The study identifies the R15L mutation in the cusS gene as a key driver of silver resistance in Escherichia coli, demonstrating that this mutation enhances the activity of the cus efflux system. Additional mutations in regulatory genes such as ompR, rho, and fur contribute to epistatic interactions that further improve silver resistance.
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