Browse AMR Genes
Explore antimicrobial resistance genes from the literature
Explore antimicrobial resistance genes from the literature
Multidrug efflux pump
Overview
Efflux-related resistance to norfloxacin, dyes, and biocides in bloodstream isolates of Staphylococcus aureus.
The study identifies several efflux pump genes (mepA, mdeA, norA, norB, norC) that are overexpressed in bloodstream isolates of Staphylococcus aureus, contributing to resistance against norfloxacin, biocides, and dyes.
Native efflux pumps contribute resistance to antimicrobials of skin and the ability of Staphylococcus aureus to colonize skin.
The study identifies tet38 and norC as efflux pumps contributing to resistance against antimicrobial fatty acids and polyamines in Staphylococcus aureus, with tet38 playing a significant role in skin colonization.
Eradication and Sensitization of Methicillin Resistant Staphylococcus aureus to Methicillin with Bioactive Extracts of Berry Pomace.
The study shows that bioactive extracts of berry pomace can sensitize methicillin-resistant Staphylococcus aureus (MRSA) to methicillin by down-regulating the expression of methicillin resistance genes (mecA) and efflux pump genes (norA, norB, norC, mdeA, sdrM, sepA).
Profiles of Staphyloccocus aureus isolated from goat persistent mastitis before and after treatment with enrofloxacin.
The study identified several AMR genes in Staphylococcus aureus isolates from goat mastitis, including blaZ, ermA, ermB, mecA, tetK, tetM, norA, norC, and lmrS. These genes conferred resistance to various antibiotics such as penicillin, erythromycin, methicillin, tetracycline, and fluoroquinolones.
Complete Genome Sequence and Analysis of a ST573 Multidrug-Resistant Methicillin-Resistant Staphylococcus aureus SauR3 Clinical Isolate from Terengganu, Malaysia.
The study identifies multiple antimicrobial resistance genes in the multidrug-resistant S. aureus isolate SauR3, including blaZ, mecA, norA, norC, sdrM, ermC, lmrS, msrA, mphC, aph(3′)-IIIa, aadE, aac(6′)Ie-aph(2″)Ia, fosB, and SAT-4, contributing to resistance against various antibiotics.
Antibiotic Susceptibility Profiling of Human Pathogenic Staphylococcus aureus Strains Using Whole Genome Sequencing and Genome-Scale Annotation Approaches.
The study identified several AMR genes in multidrug-resistant S. aureus strains, including blaZ, mecA, mecC, norA, norC, MgrA, tet(45), APH(3′)-IIIa, ermC, AAC(6′)-APH(2″), and fusC. These genes confer resistance to various antibiotics such as beta-lactams, fluoroquinolones, tetracyclines, aminoglycosides, macrolides, lincosamides, streptogramin B, and fusidic acid.
Occurrence of antimicrobial-resistant Staphylococcus aureus in a Brazilian veterinary hospital environment.
The study identified several AMR genes in Staphylococcus aureus isolates from a Brazilian veterinary hospital, including blaZ, mecA, norA, norC, tetM, tet38, ermA, and ermB, which confer resistance to various antibiotics such as penicillin, methicillin, tetracycline, and erythromycin.
Genomic alterations involved in fluoroquinolone resistance development in Staphylococcus aureus.
The study identifies mutations in grlA (S80F) and gyrB (T451S and/or R450S) as key contributors to fluoroquinolone resistance in Staphylococcus aureus, along with overexpression of rimI and fmtB.
Genetic and Phenotypic Characterization of Subclinical Mastitis-Causing Multidrug-Resistant Staphylococcus aureus.
The study identified blaZ, norA, norC, tet38, and icaD genes in multidrug-resistant Staphylococcus aureus isolates associated with subclinical mastitis. These genes were linked to resistance against penicillin, tetracycline, macrolides, fluoroquinolones, and biofilm formation.
Multidrug Efflux System-mediated resistance in Staphylococcus aureus under a One Health approach.
The study identifies several multidrug efflux system genes (norA, norB, norC, lmrS, tet38, msrA) in Staphylococcus aureus isolates from human, animal, and food sources, highlighting their role in resistance to various antibiotics.
Resistome, mobilome, and virulome explored in clinical isolates derived from acne patients in Egypt: unveiling unique traits of an emerging coagulase-negative Staphylococcus pathogen.
The study identified multiple antibiotic resistance genes in coagulase-negative staphylococci (CoNS) isolates from acne patients in Egypt, including blaZ, mecA, tet(K), erm(C), lnuA, vgaA, dfrC, fusB, fosBx1, norA, and vanT. These genes were found to be located on plasmids and chromosomes, indicating a multidrug-resistant profile.
Deciphering the genomic character of the multidrug-resistant Staphylococcus aureus from Dhaka, Bangladesh.
The study identified multiple AMR genes in multidrug-resistant S. aureus isolates from Bangladesh, including mecA, blaZ, ermC, mepA, norA, norC, sdrM, and lmrS, which confer resistance to various antibiotics.
A Mechanistic Insight into the Anti-Staphylococcal Mode of Action of (+)-Usnic Acid and Its Synergy with Norfloxacin Against Methicillin-Resistant Staphylococcus aureus.
The study demonstrates that (+)-Usnic acid (UA) exhibits strong anti-staphylococcal activity and synergizes with Norfloxacin (NOR) against methicillin-resistant Staphylococcus aureus (MRSA). UA reduces the minimum inhibitory concentrations (MICs) of various antibiotics, including Oxacillin, Cefoxitin, Cefazolin, Ciprofloxacin, Tetracycline, Erythromycin, Streptomycin, and Vancomycin. The combination of UA and NOR shows synergistic interactions against all MRSA clinical isolates.
Comparative genomics of endemic Staphylococcus aureus ST1 in New Zealand.
The study identified several AMR genes in Staphylococcus aureus ST1 isolates from New Zealand, including blaZ, tet(38), mecA, mecR1, qacA, qacB, ermC, fusC, mupA, and others. These genes were associated with resistance to various antibiotics such as penicillin, tetracycline, methicillin, quaternary ammonium compounds, erythromycin, fusidic acid, mupirocin, and fluoroquinolones. The study also highlighted the presence of a novel prophage, φSabovST1, in bovine ST1 isolates, which encoded bovine-adapted leukocidins.
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