Browse AMR Genes
Explore antimicrobial resistance genes from the literature
Explore antimicrobial resistance genes from the literature
Overview
| Protein Change | Nucleotide Change | Mechanism | Organism | Resistance To | Database | Validation Status |
|---|---|---|---|---|---|---|
| V606A | - | - | Escherichia coli | Aztreonam|Clavulanic acid | Reslit | Candidate |
| R100H | - | confirmed that both mutations have to present to obtain high level resistance | Salmonella Typhimurium | Triclosan | Reslit | Candidate |
| R147S | - | negatively regulates 2,4-DAPG and Plt biosynthesis by binding to promoter regions of phlA, phlG, pltR, pltL, and pltF | Pseudomonas protegens | - |
| Candidate |
| I128N | - | - | Escherichia coli | Trimethoprim | Reslit | Candidate |
| N98H | - | - | Salmonella enterica | Chloramphenicol|Tetracycline|Fluoroquinolones | Reslit | Candidate |
| T298I | - | Escherichia coli | Fluoroquinolones | Reslit | Candidate |
The impact of aztreonam-clavulanic acid exposure on gene expression and mutant selection using a multidrug-resistant E. coli.
plays a crucial role in bacterial stress response
Importance of sigma factor mutations in increased triclosan resistance in Salmonella Typhimurium.
Regulatory roles of RpoS in the biosynthesis of antibiotics 2,4-diacetyphloroglucinol and pyoluteorin of Pseudomonas protegens FD6.
RpoS wild-type negatively regulates 2,4-DAPG and Plt biosynthesis by binding to promoter regions of phlA, phlG, pltR, pltL, and pltF.
Pervasive gene deregulation underlies adaptation and maladaptation in trimethoprim-resistant E. coli.
rpoS loss-of-function mutations alleviate fitness costs of mgrB deficiency and contribute to trimethoprim resistance.
Long-term exposure to food-grade disinfectants causes cross-resistance to antibiotics in Salmonella enterica serovar Typhimurium strains with different antibiograms and sequence types.
Evolutionary Instability of Collateral Susceptibility Networks in Ciprofloxacin-Resistant Clinical Escherichia coli Strains.
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