Browse AMR Genes
Explore antimicrobial resistance genes from the literature
Explore antimicrobial resistance genes from the literature
Overview
| Protein Change | Nucleotide Change | Mechanism | Organism | Resistance To | Database | Validation Status |
|---|---|---|---|---|---|---|
| K88E | - | Target modification | Campylobacter jejuni | Streptomycin | ResFinder | Established |
| K121K | - | - | Mycobacterium tuberculosis | streptomycin | Reslit | Candidate |
| K42N | - | impairs ribosome performance | Escherichia coli, Mycobacterium tuberculosis, Salmonella enterica | streptomycin |
Reslit |
| Candidate |
| P90S | - | impairs ribosome performance | Salmonella enterica, Escherichia coli, Mycobacterium tuberculosis | streptomycin | Reslit | Candidate |
| K42R | - | retains wild-type translation rate | Salmonella enterica, Escherichia coli, Mycobacterium tuberculosis | streptomycin | Reslit | Candidate |
| K88R | - | Target modification | Campylobacter jejuni | Streptomycin | ResFinder | Established |
| K43R | Target modification | Campylobacter jejuni, Mycobacterium tuberculosis | Streptomycinstreptomycin | ResFinderReslit | Confirmed |
| K88Q | - | Target modification | Campylobacter jejuni | Streptomycin | ResFinder | Established |
| - | - | E. coli | kanamycin | Reslit | Candidate |
Mutations in the rpsL gene are involved in streptomycin resistance in Campylobacter coli.
Molecular characterization of multidrug-resistant isolates of Mycobacterium tuberculosis from patients in North India.
The fitness cost of streptomycin resistance depends on rpsL mutation, carbon source and RpoS (σS).
Mutations in rpsL are responsible for streptomycin resistance and have varying effects on bacterial fitness depending on environmental conditions.
Single ribosomal protein mutations in antibiotic-resistant bacteria analyzed by mass spectrometry.
Fluoromycobacteriophages for rapid, specific, and sensitive antibiotic susceptibility testing of Mycobacterium tuberculosis.
Sublethal antibiotic treatment leads to multidrug resistance via radical-induced mutagenesis.
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