Browse AMR Genes
Explore antimicrobial resistance genes from the literature
Explore antimicrobial resistance genes from the literature
D-alanine--D-serine ligase VanL
Overview
| Allele | Database | Papers | Drug Classes | Organisms | Countries | Years | Sequence Accession | Protein Accession |
|---|---|---|---|---|---|---|---|---|
| VanL | Card DatabaseReference Gene CatalogReslit | 8 | VANCOMYCIN, Vancomycin +1 | Enterococcus faecalis +5 | Global, Belgium, China, China|Xinjiang | 2008, 2022, 2023, 2024, 2025 |
| EU250284.1 |
| ABX54687.1 |
| vanL-like | Reslit | 1 | Glycopeptide | Chryseobacterium oranimense G311 | France | 2015 | CDHM01000001|CDHM01000002|CDHM01000003|CDHM01000004|CDHM01000005|CDHM01000006|CDHM01000007|CDHM01000008|CDHM01000009|CDHM01000010|CDHM01000011|CDHM01000012|CDHM01000013|CDHM01000014|CDHM01000015 | - |
| vanL | Card Database | 1 | - | Enterococcus faecalis | - | - | EU250284.1 | ABX54687.1 |
Molecular characterization of Enterococcus faecalis N06-0364 with low-level vancomycin resistance harboring a novel D-Ala-D-Ser gene cluster, vanL.
Molecular characterization of Enterococcus faecalis N06-0364 with low-level vancomycin resistance harboring a novel D-Ala-D-Ser gene cluster, vanL.
Molecular characterization of Enterococcus faecalis N06-0364 with low-level vancomycin resistance harboring a novel d-Ala-d-Ser gene cluster, vanL.
The study identifies a novel d-Ala-d-Ser gene cluster, vanL, responsible for low-level vancomycin resistance in Enterococcus faecalis N06-0364.
Whole-genome sequence of Chryseobacterium oranimense, a colistin-resistant bacterium isolated from a cystic fibrosis patient in France.
The study identified multiple AMR genes and mutations in Chryseobacterium oranimense G311, a colistin-resistant bacterium isolated from a cystic fibrosis patient. These include various beta-lactamases, aminoglycoside modifying enzymes, tetracycline resistance genes, MLS resistance genes, phenicol resistance genes, glycopeptide resistance genes, fluoroquinolone resistance genes, sulfonamide resistance genes, rifampin resistance genes, and multidrug efflux pumps. Additionally, mutations in pmrA, pmrB, and lpxA were found to contribute to colistin resistance.
The resistomes of Mycobacteroides abscessus complex and their possible acquisition from horizontal gene transfer.
The study identifies numerous AMR genes in Mycobacteroides abscessus complex, highlighting the widespread presence of resistance to multiple antibiotic classes, including beta-lactams, aminoglycosides, glycopeptides, and others. Key findings include the detection of beta-lactamases like blaLAP-1 and blaTLA-2, 23S rRNA methyltransferases such as erm(33), erm(43), and erm(44), and various aminoglycoside modifying enzymes. Additionally, vancomycin resistance genes like vanA, vanB, and vanC were identified, along with efflux pump genes contributing to multidrug resistance.
Vancomycin Resistance in Enterococcus and Staphylococcus aureus.
The paper discusses the genetic basis of antibiotic resistance mechanisms in Enterococcus and Staphylococcus aureus, focusing on various resistance genes and mutations associated with glycopeptides, aminoglycosides, beta-lactams, macrolides, lincosamides, streptogramins, quinolones, tetracyclines, and fosfomycin.
An in-house 45-plex array for the detection of antimicrobial resistance genes in Gram-positive bacteria.
The study describes an in-house 45-plex array for detecting antimicrobial resistance genes in Gram-positive bacteria, identifying optrA, poxtA, and vanA as significant resistance markers in Enterococcus and Staphylococcus isolates.
Resistance phenotype and genetic features of a heterogeneous vancomycin intermediate-resistant Staphylococcus aureus strain from an immunocompromised patient.
The study characterizes a heterogeneous vancomycin intermediate-resistant Staphylococcus aureus (hVISA) strain, identifying multiple mutations in genes associated with vancomycin resistance, including fmtB, mprF, dltA, dltD, lytM, pbp4, sceD, tagA, graR, vraG, vraR, yycH, yycI, mutL, rpoD, sigB, msaC, trpC, and atp.
Virulence and resistance gene analysis of Rothia nasimurium by whole gene sequencing.
The study identified multiple AMR genes in Rothia nasimurium Y1, including vanA, vanC, vanB, vanE, vanD, vanG, vanF, vanM, vanL, vanO, vanN, mtrA, vanRA, arlR, vanRI, vanRB, vanRC, vanRD, vanRF, vanRG, CpxR, kdpE, vanRM, vanRN, baeR, adeR, vanRL, smeR, gyrA, gyrB, parC, Mfd, mfd, PBP2, PBP2x, EF-Tu, dfrE, pncA, tetB(P), tetQ, tet44, tetT, tetW, tetS, tetM, tetO, otr(A), tet36, tet32, clbC, clbB, clbA, cipA, cfrA, cfrC, sul3, ParY, murA, cls, and ileS, which confer resistance to various antibiotics such as glycopeptides, beta-lactams, fluoroquinolones, tetracyclines, sulfonamides, aminoglycosides, lincosamides, phenicols, macrolides, and others.
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