Browse AMR Mutations
Explore antimicrobial resistance mutations from the literature
Explore antimicrobial resistance mutations from the literature
Overview
Mutation rate and evolution of fluoroquinolone resistance in Escherichia coli isolates from patients with urinary tract infections.
Genetic regulation of the ramA locus and its expression in clinical isolates of Klebsiella pneumoniae.
Mutations in acrR were found to lead to AcrAB overexpression, contributing to tigecycline resistance.
Interactions of Polymyxin B in Combination with Aztreonam, Minocycline, Meropenem, and Rifampin against Escherichia coli Producing NDM and OXA-48-Group Carbapenemases.
associated with increased AcrAB-TolC efflux activity
Tigecycline Nonsusceptibility Occurs Exclusively in Fluoroquinolone-Resistant Escherichia coli Clinical Isolates, Including the Major Multidrug-Resistant Lineages O25b:H4-ST131-H30R and O1-ST648.
Mutations in acrR contribute to tigecycline nonsusceptibility by overexpression of AcrAB-TolC.
The effect of commonly used non-antibiotic medications on antimicrobial resistance development in Escherichia coli.
Mutation in AcrR leads to overexpression of the AcrAB-TolC efflux pump, contributing to ciprofloxacin resistance.
The T5N mutation in AcrR leads to overexpression of the AcrAB-TolC efflux pump, contributing to ciprofloxacin resistance.
Genetic characterization of highly fluoroquinolone-resistant clinical Escherichia coli strains from China: role of acrR mutations.
acrR
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