Browse AMR Mutations
Explore antimicrobial resistance mutations from the literature
Explore antimicrobial resistance mutations from the literature
Overview
Multidrug-resistant tuberculosis clusters and transmission in Taiwan: a population-based cohort study.
These mutations are associated with rifampicin resistance and are compensatory mutations.
Characterization of rifampicin-resistant Mycobacterium tuberculosis in Khyber Pakhtunkhwa, Pakistan.
Characteristics of compensatory mutations in rifampicin-resistant tuberculosis and their association with compensated transmission in Ningbo, China.
putative compensatory mutation
Genomic characterization and epidemiology of Mycobacterium tuberculosis lineage 2 isolates from Kazakhstan.
Large-scale statistical analysis of Mycobacterium tuberculosis genome sequences identifies compensatory mutations associated with multi-drug resistance.
These mutations are associated with rifampicin resistance.
Fitness costs of rifampicin resistance in Mycobacterium tuberculosis are amplified under conditions of nutrient starvation and compensated by mutation in the β' subunit of RNA polymerase.
Mutations in rpoC are compensatory for rpoB mutations
A complete high-quality MinION nanopore assembly of an extensively drug-resistant Mycobacterium tuberculosis Beijing lineage strain identifies novel variation in repetitive PE/PPE gene regions.
Transmission of multidrug-resistant tuberculosis in Beijing, China: An epidemiological and genomic analysis.
rpoC V483G and rpoC V483A were the most frequent mutations.
Multi-clonal evolution of multi-drug-resistant/extensively drug-resistant Mycobacterium tuberculosis in a high-prevalence setting of Papua New Guinea for over three decades.
Mutations promoted from gene entry 'rpoC' (model emitted mutations_found inside genes).
Source of the Fitness Defect in Rifamycin-Resistant Mycobacterium tuberculosis RNA Polymerase and the Mechanism of Compensation by Mutations in the β' Subunit.
The β′F452L and β′V483G mutations are compensatory mutations that mitigate the fitness defects of the βS450L mutation.
Exploiting Homoplasy in Genome-Wide Association Studies to Enhance Identification of Antibiotic-Resistance Mutations in Bacterial Genomes.
Mutation in rpoC leads to resistance to rifampicin.
Integrating standardized whole genome sequence analysis with a global Mycobacterium tuberculosis antibiotic resistance knowledgebase.
Mycobacterium tuberculosis rpoC mutations confer resistance to rifampicin; CARD accession: CARD:3004994
Genome-wide analysis of multi- and extensively drug-resistant Mycobacterium tuberculosis.
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